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doi: 10.1016/j.intimp.2020.106328 236 WangXGongSPuDHuNWangYFanPet al

CYP2D6 may also contribute to the formation of NAPQI, albeit to a lesser extent than other P450 isozymes, and its activity may contribute to paracetamol toxicity, in particular, in extensive and ultrarapid metabolizers and when paracetamol is taken at very large doses.[18] The metabolism of paracetamol is an excellent example of toxication, because the metabolite NAPQI is primarily responsible for toxicity rather than paracetamol itself

Studies in which these cytokines were blocked or absent showed significantly impaired fracture healing

But most of this research is co-authored by Sikiric, who holds several patents related to BPC-157, and some experts have accused him of cherry-picking evidence