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We have demonstrated the ability to deliver multi-milligram weekly doses of a GLP-1 with a patch, and reach the exposures obesity treatment requires, without an injection or cold storage

[3] However, the precise contribution of each receptor pathway to gastrointestinal tolerability in a triple agonist has not been fully characterised in humans, and this attribution should be regarded as informed by class-effect data rather than confirmed mechanism

This stands in stark contrast to clinical trials that aimed to reduce the level of beta-amyloid in the brain, where only limited improvements have been observed that are not considered to be clinically meaningful [1, 4]

Mitochondrial generation of superoxide and hydrogen peroxide as the source of mitochondrial redox signaling

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