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Demonstrating peripheral effects by suppressing T-cell-mediated inflammation via GLP-1 receptor activation in intestinal lymphocytes, and central effects by mediating Toll-like receptor (TLR)-induced anti-inflammatory responses through GLP-1 receptor signaling in the brain [39]

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Dual role of NO donors in the reversal of tumor cell resistance and EMT: Downregulation of the NF-B/Snail/YY1/RKIP circuitry

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