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BPC-157 has been shown in animal studies to adhere to the metabolic process of standard peptide-based drugs, suggesting that injections are the most bioavailable route of BPC-157 administration [30, 31]

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The results indicated that pretreatment with 10 M ERK1/2 inhibitor obviously antagonized, while pretreatment with 10 M JNK inhibitor and 10 M p38 inhibitor had no effect on, BPC-157-induced proliferation, migration, and tube formation

As a single-center, retrospective study, our findings may be subject to referral bias and incomplete data capture

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