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doi:10.1016/j.jtherbio.2022.103376

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18 In kidney injury, extracellular vesicles and secreted lipid mediators are sufficient to synchronize ferroptotic death across spatially contiguous tubular epithelial cells, thereby contributing to a self-amplifying wave of tubular death during the transition from acute kidney injury to chronic kidney disease

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Research published in peer-reviewed journals has documented BPC-157's capacity to upregulate the expression of vascular endothelial growth factor (VEGF) receptors and promote endothelial cell migration in vitro