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Furthermore, we show that p53 phosphorylation enhances the affinity for both FOXO4 and FOXO4-DRI
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Wound healing studies in human subjects document accelerated re-epithelialization, reduced inflammatory markers (IL-1, TNF-), and improved tissue tensile strength in GHK-Cu treated wounds compared to controls
GLP-1 agonists can reduce plasminogen activator inhibitor (PAI)1 levels, thereby augmenting fibrinolytic potential [96, 97]
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