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For the L-GSH supplementation group, the levels of reduced form of GSH remained relatively the same and were not significantly different between pre- and post- L-GSH supplementation in plasma ( Figure 4F ) and PBMCs ( Figure 4G )

Further mechanistic investigations determined that the elevation of intracellular GSSG levels resulted in NF-B inhibition by facilitating glutathionylated IKK- (IKK--SSG) formation, leading to enhanced TNF hepatotoxicity

Gene expression profiling of major depression and suicide in the prefrontal cortex of postmortem brains

doi: 10.1007/s12031-025-02384-x 214 ChenLMinJWangF