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It binds pituitary GHRH receptors to drive a pulsatile release of growth hormone , which in turn raises IGF-1

M., G130V, a common FRDA point mutation, appears to have arisen from a common founder

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The broader picture is one in which MOTS-c addresses a gap that many other longevity interventions leave unfilled: the mitochondrial signaling decline that is simultaneously a cause and consequence of metabolic aging

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