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After fixation, the cells were washed with PBS and permeabilized with 0.1% Triton X-100 (Daejung 8566-4405) in PBS for 10 min at room temperature
Collectively, these data again suggest that GIPR antagonism, unlike agonism, mimics GLP-1R agonism in the DVC and that GIPR, unlike GLP-1R, is the primary target for MAR709 in the DVC
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The alkyl radical in the presence of oxygen is converted to peroxyl radicals which facilitate the long-term damage by continually regenerating themselves through hydrogen abstraction from the nearby biomolecules and forming the hydroperoxides as the byproduct (Fig
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