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Through this mechanism, GLP-1RAs may reduce endothelial dysfunction and oxidative stress, both of which are key contributors to sepsis pathophysiology (57)
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Concerns have been raised regarding the potential for abnormal cell growth, particularly in the absence of long-term safety data from clinical research
The first and most relevant to the discussion of inflammatory immune responses is Nuclear Factor kappa-light-chain-enhancer of activated B cells, or NF-kB for simplicity
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