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Proc Natl Acad Sci U S A , 87:71857189

Treatments for keloids remain unsatisfactory because the key pathophysiological mechanism is unknown 1

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Recent lichen-focused syntheses further emphasize that secondary metabolite classes characteristic of lichens, such as depsides, depsidones, and usnic-acid derivatives, may interfere with membrane integrity, urease-associated virulence, and biofilm-related pathways, thereby contributing to multi target antimicrobial profiles (Tian et al

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