Lipid ROS, oxidized phospholipids, and malondialdehyde are frequently used as evidence of propagated ferroptosis, yet none of these markers is uniquely specific to ferroptotic death
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Ismail et al., 2009), hypometabolism in the anterior cingulate cortex with appetite disturbances (Hu et al., 2002), and atrophy of the cingulate and frontal gyri with disinhibition (Serra et al., 2010)
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