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Malaguarnera, M., Gargante, M

Mitochondrial dysfunction leading to oxidative stress also induces FGF21 expression in preclinical disease models ( Enhanced secretory load, imbalances in redox homeostasis, or perturbations in protein homeostasis triggered by lysosomal or autophagosomal dysfunction may all cause ER stress and trigger the unfolded protein response (UPR)

Platelet-rich plasma has been widely investigated as a regenerative therapy in I/R injury across different organ systems

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Therapies aimed at restoring a healthy microbiome or blocking deleterious MGBA signals (like LPS or certain bile acids) are being explored as novel ways to slow AD progression

These results suggest that Mcp1 plays an important role in the maintenance of mitochondrial function and morphology in C