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For DSIP with no identified receptor and no established axis the concept has no mechanistic anchor

Although the factors that promote this dysregulation are poorly understood in PD, recent data suggest that increased -synuclein levels can reduce autophagic flux by decreasing the autophagosomal membrane-associated protein LC3, resulting in further accumulation of -synuclein and cytotoxicity (Figure 3A) (Lei et al., 2019)

Personally, weve had people who had to take a very, very small dose of just 50mg every other day until they worked with one of our dietitians to open up their pathways and clear toxins before they could use a traditional dose of 500mg