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In animal models of acute lung injury induced by lipopolysaccharide (LPS), GHK-Cu treatment reduced reactive oxygen species (ROS) production and increased superoxide dismutase (SOD) activity while decreasing TNF- and IL-6 production

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Postnatal adaptation was uneventful

Weight outcomes included the absolute and percent changes in weight from the index date to 1 year of post-index follow-up (index date + 365 days)

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