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Zhang, W., Miao, J., Zhang, G., Liu, R., Zhang, D., Wan, Q., Yu, Y., Zhao, G., and Li, Z

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doi:10.1007/s11011-012-9324-8, PMID: 22718491 Morris G, Maes M (2012) Increased nuclear factor-B and loss of p53 are key mechanisms in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS)

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Tissue-Repair & Regenerative Research Research involving GHK-Cu commonly investigates: Tissue-remodeling pathways Recovery-focused regenerative signaling Fibroblast-related mechanisms Keratinocyte-related pathways Connective-tissue and extracellular-matrix signaling Scar-remodeling and tissue-maintenance research These pathways have contributed to broader research interest in GHK-Cu within regenerative and recovery-focused research models