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p53-dependent chemokine production by senescent tumor cells supports NKG2D-dependent tumor elimination by natural killer cells
This cycle reinforces itself over time
Blair JMA, Bavro VN, Ricci V et al (2015) AcrB drug-binding pocket substitution confers clinically relevant resistance and altered substrate specificity
The elimination strategy targeting senescent cells thus represents a promising area of research in the context of various pathologies, including cancer, cardiovascular, and neurodegenerative diseases, by targeting the very biological foundation of aging
Somatostatin is a bit of a break or an antagonist on growth hormone release
You can perform more repetitions per set instead and get similar benefits
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