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Similar clinical symptoms are found in individuals with hereditary IF deficiency (also called congenital pernicious anemia) in whom the lack of IF results in the defective absorption of vitamin B 12
Rev1 contributes to proper mitochondrial function via the PARP-NAD(+)-SIRT1-PGC1 axis
Hum Reprod
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A vial at day 21 has not suddenly become useless
The evidence is preclinical (mouse and cell studies)
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