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Both animal and human studies demonstrate that LPS stimulate GLP-1 release via Toll-like receptors (TLRs)

Despite the extensive evidence supporting the beneficial effects of GLP-1RAs on the central nervous system (CNS), emerging studies have raised concerns about potential adverse effects

A recent report showed that HK-2-mediated H3K18la enrichment at the promoter of the Acta2 gene in TGF-1-induced hepatic stellate cells (HSCs) was a prerequisite for HSC activation and liver fibrosis [29]

This interplay suggests that DNA damage, stemming from phospholipid peroxidation-induced iron deposition, might be a primary instigator

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It was meant to rule out deficiency, but the result points the other direction