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One hypothesis for this change is related to neuroinflammation markers and to microvascular lesions involved in Alzheimers disease
doi:10.1590/0001-3765202020191486 Otero JS, Hirsch GE, Klafke JZ, et al
We subsequently concentrated on the subset of proteins enriched in pulldowns from senescent cells when compared with non-senescent controls
Without this enzyme, GSH would not be able to perform its crucial role in maintaining the body's oxidative balance
Kei EF et al
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