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Furthermore, many studies focus on its potential role in tissue repair and regeneration research

No formal human pharmacokinetic study has been published

Broader solid tumor studies corroborate that pharmacological inhibition of CAF-mediated Gln synthesis disrupts tumor metabolic fitness, suggesting combinatorial targeting of tumoral and stromal glutamine metabolism, particularly through dual GLS1/ECM pathway inhibition, may offer promising therapeutic avenues for advanced prostate cancer (196)

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(2021) reported that a GLUT1-targeted nanodisk underwent endocytosis by clathrin-mediated endocytosis and energy-dependent macro-pinocytosis pathway (84)