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Takahashi, M

However, exclusive use of folic acid in treating vitamin B12 deficient megaloblastic anemia could result in progressive and irreversible neurologic damage

Potential mechanisms being explored include: Reduction in pro-inflammatory cytokines Antioxidant enzyme support Reduction in oxidative tissue damage Improved tissue healing environment This is one reason GHK-Cu has become so popular in regenerative and longevity medicine

secretory IgA may be mildly reduced

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In functional medicine, BPC-157 is often combined with other peptides (like TB-500 or Thymosin-1) or detox and support protocols to create a synergistic healing effect