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While GHK alone has some biological activity, it is the GHK-Cu form that is responsible for the vast majority of the regenerative, anti-inflammatory, and gene-modulating effects discussed in clinical research
This distinction is crucial: most fundamental studies on the actin mechanism focus on the entire T4, and extrapolation to the TB-500 fragment alone remains incomplete in the published literature
Journal of molecular medicine (Berlin, Germany), 95 (3), 323333
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