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GLP-1 receptor agonists enhance glucose-dependent insulin secretion from pancreatic beta cells while suppressing inappropriate glucagon release, thereby improving glycemic control

Although data in kidney transplant recipients (KTR) remain limited, the high prevalences of overweight and obesity, post-transplant diabetes mellitus, and cardiovascular disease in this population make GLP-1RAs a promising therapeutic strategy

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Structural basis for delivery of the intact [Fe2S2] cluster by monothiol glutaredoxin