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The glucose-dependent insulinotropic polypeptide receptor (GIPR) functions similarly but may also be expressed in - and possibly -cells, modulating glucagon and somatostatin secretion (Shilleh et al., 2024)
That extended activity window changes everything about how the peptide works in the body
While a causal relationship in humans has not been established, clinical vigilance is advised
This response typically stems from rapid metabolic shifts, dehydration, and changes in blood sugar regulation as your body adjusts to GLP-1 therapy
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