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Since an IGF-1R construct, in which the tyrosine at position 1136 was replaced by a phenylalanine, also led to a strong inhibition of phosphorylated Akt in transfected cells, it was suggested that this mechanism may be responsible for the apoptotic effect of PPP (Vasilcanu et al, 2004)

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Lipoic promotes anti-inflammatory and antithrombotic (blood clot-reducing) pathways and increases nitric oxide-mediated vasodilatation (blood vessel widening) [20]

One of the standout trends fueling this shift is peptide therapy

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