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[7] Protein folding studies on Thioredoxin revealed that a minimum peptide length of 83 residues is required to acquire secondary and tertiary structure as shown by Ghosal et.al in 1999

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Furthermore, both SM and exenatide selectively activated p38 MAPK phosphorylation in the spinal cord, providing additional evidence that SM exerts its anti-allodynic effects by targeting spinal GLP-1R and promoting microglial -endorphin synthesis via the p38 MAPK signaling cascade [72]

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