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The primary pathway is glucuronidation via UGT1A enzymes, which accounts for roughly 90% of clearance [1]
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The short half-life (12 min in humans) results from dipeptidylpeptidase-IV (DPP-IV) proteolysis by which the N-terminal dipeptide is rapidly removed, inactivating GLP-1
This implies these findings may not generalize to a more diverse population or other conditions
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