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There is strong evidence that hyperglycemia results in the activation of PKC in diabetic glomeruli and, as a consequence, mesangial expansion, glomerular basement membrane thickening, endothelial cell dysfunction leading to diabetic renal disease, inflammation, apoptosis [48, 49, 50]

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Derivatives of clinical novel inhibitors Despite the advances made in GLS inhibitors, the development of precursor drugs has encountered significant setbacks due to a lack of selectivity and low bioavailability