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These medicines work on brain reward pathways and satiety signals, potentially reducing food cravings and binge episodes

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Ish-Shalom D et al

Accessed August 2024 at Nadkarni P, Chepurny OG, Holz GG

These observations strongly support the current hypothesis that reactive metabolite formation and protein binding especially in mitochondria are the key initiating events in the toxicity, which is then the trigger for a moderate mitochondrial oxidant stress with release of superoxide by complex III towards the cytosol without compromise of mitochondrial respiration [33]