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The transcription factor p53 is a vital regulator that translates the occurrence of genotoxic stress into alterations of gene expression (Tishler et al., 1993)

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Published scientific literature discusses GHK-Cu in relation to copper peptide research, peptide chemistry, gene expression studies, molecular biology, receptor biology, cellular communication, and analytical peptide characterization

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Under selenium deficiency, the protein expressions and/or activities of seleno-GPxs are decreased in a hierarchical order depending on their importance for cellular viability and functions (Table 1) [8, 25]