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SGLT2 inhibitors reduce hyperfiltration through the above mentioned mechanism, and attenuate/prevent the molecular markers of kidney growth, fibrotic responses of proximal tubular cells and glomerular size, as well as gluconeogenesis in diabetic Akita rats [19, 20]

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The glyoxalase pathway (consisting of glyoxalase I and glyoxalase II enzymes) for detoxification of methylglyoxal, a cytotoxic molecule, also requires GSH in the first reaction step

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