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In tumor systems, for example, galectin-13 (LGALS13) released by ferroptotic cells does not appear to directly convey oxidative species to neighboring cells

Hexarelin, a novel GHRP-6 analog, stimulates growth hormone (GH) release in a GH-secreting rat cell line (GH1) insensitive to GH-releasing hormone

Lowering GSH levels also triggers ferroptosis through deactivation of GPX4, leading to accumulation of lipid peroxides [87] In some cases, GSH depletion alone can induce therapeutic effects without relying on Fenton chemistry, which typically involves HO reacting with Fe 2 to form hydroxyl radicals (OH) that damage critical biomolecules such as DNA, proteins, and lipids

In this sense, MOTS-c works as an endogenous exercise mimetic, triggering AMPK activation through the same molecular lever that exercise pulls, just from the inside [1]

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