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Elevated ROS levels are a hallmark of leukemia cells, resulting from molecular mechanisms involving NADPH oxidase, the mitochondrial electron transport chain (mtETC), and cytochrome P450 [34, 90]

Even so, moving from this biochemical function to clinical outcomes is not always straightforward: the evidence can be heterogeneous and depends on dose, duration, the population studied and lifestyle variables

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Regulation of the OxyR transcription factor by hydrogen peroxide and the cellular thiol-disulfide status

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