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Studies in humans and animals with non-alcoholic fatty liver disease show that CD44 plays a key role in the inflammation and liver damage in NAFLD.[ref] Other genetic studies have looked at genes in pathways known to be involved in acetaminophen metabolism, including genes related to sulfation, glucuronidation, and glutathione production

Establishing comprehensive ethical and legal frameworks, developing animal-free culture media, and exploring alternative cell sources are essential prerequisites to advancing the clinical translation of kidney organoid technologies

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