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Animal models show this peptide induces concentration-dependent vasodilation, an effect that was diminished when NO synthesis was inhibited
Binding/competition metrics in receptor systems (e.g., IC50/Ki where applicable) Second-messenger readouts (assay-dependent) Pathway readouts (e.g., phosphorylation markers) in controlled cell models (assay-dependent) Receptor trafficking/internalization (imaging or tagged-receptor assays) Stability/integrity profiling (e.g., HPLC profiles
This homeostatic modulation, rather than directional stimulation, suggests a fundamentally different mechanism than most vasodilators
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