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AA binds to PPAR-2 to stimulate GLUT4 expression in HepG2 cell line, exhibiting an alternative insulin-independent activation of GLUT4 [157]

Abstract Lipid storage myopathies are considered inborn errors of metabolism affecting the fatty acid metabolism and leading to accumulation of lipid droplets in the cytoplasm of muscle fibers

Following the rate determining formation of 8-enolate , this intermediate shows only marginal stabilization over the corresponding transition state and evolves to 8-B4 in a barrierless step

Figure 2A depicts a group of TH-positive cells that are negative for neuromelanin

Michelotti, A